Even before SCD was developed,some brilliant celiac experts realized that fat and protein absorption improved when carbohydrates were excluded from the diet. This happens because the gut pathogens that are eating the undigested carbs will starve when there is no food for them. When the gut pathogens disappear,the damage to the GI tract stops and the gut can heal; fat and protein can then be absorbed.

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To learn more about how carbohydrate malabsorption leads to impaired fat absorption read the Celiac Story in Breaking The Vicious Cycle.

http://www.scdiet.org/7archives/scdceli1.html

Celiac pioneer,Dr Howland, stated the following: "From clinical experience, it has been found that of all the elements of food, carbohydrate is the one which must be excluded rigorously; that with this greatly reduced, the protein and fat are almost always well digested even though the absorption of fat may not be as satisfactory as in health."

This was written before SCD was developed,there are better results for fat malabsorption with SCD

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There are studies that support the concept that severe malabsorption of dietary fats and proteins play a primary causative role in enteric hyperoxaluria. Since SCD heals the gut and improves the body's ability to absorb fats and protein,it is important to pay attention to the role of fat absorption in the formation of hyperoxaluria.

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In one experiment, rats were given orlistat,a gastrointestinal lipase inhibitor to prevent them from absorbing fats. The rats who had not been able to absorb fat had a significant increase of urinary oxalate.

These findings support the theory that unabsorbed fat and bile acids may react with calcium in the intestinal lumen, limiting the amount of free calcium binding with oxalate and thereby raising intestinal oxalate absorption leading to hyperoxaluria.

The results of these studes support the concept that severe malabsorption of dietary fat plays a primary causative role in enteric hyperoxaluria.

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http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=pubmed
&dopt=Abstract&list_uids=15253722&query_hl=3

1: Kidney Int. 2004 Aug;66(2):676-82. Related Articles, Links

Erratum in:

* Kidney Int. 2004 Sep;66(3):1304. Heiberg, Ita Pfeferman [corrected to Heilberg, Ita Pfeferman].

Fat malabsorption induced by gastrointestinal lipase inhibitor leads to an increase in urinary oxalate excretion.

Ferraz RR, Tiselius HG, Heilberg IP.

Nephrology Division, Universidade Federal de Sao Paulo, UNIFESP, Sao Paulo, Brazil.

BACKGROUND: Unabsorbed fat and bile acids may react with calcium in the intestinal lumen, limiting the amount of free calcium binding with oxalate and thereby raising intestinal oxalate absorption leading to hyperoxaluria. The aim of the present study was to determine whether orlistat (Xenical), a gastrointestinal lipase inhibitor, might increase urinary oxalate in an experimental rat model. METHODS: Thirty-nine male adult Wistar rats were fed a standard diet alone (controls) or supplemented with either 2% sodium oxalate (NaOx) or 3.2 mL of soy oil, or with both (NaOx + soy oil) for 4 weeks (diet period). Orlistat (16 mg/day) was added to the diet from the 5th to the 8th week (diet + orlistat period). Urinary oxalate (uOx), calcium (uCa), magnesium (uMg), and citrate (uCit) were determined and the ion-activity product of calcium oxalate [AP (CaOx) index(rat)] was estimated. RESULTS: Compared to baseline uOx significantly increased after diet + orlistat in controls (0.64 +/- 0.1 mg/24 hours vs. 0.56 +/-0.1 mg/24 hours), soy oil (0.80 +/- 0.3 mg/24 hours vs. 0.49 +/-0.2 mg/24 hours), and NaOx (2.48 +/- 0.8 mg/24 hours vs. 0.57 +/- 0.2 mg/24 hours), but the most marked increase occurred in NaOx + soy oil (3.87 +/- 0.7 mg/24 hours vs. 0.47 +/- 0.1 mg/24 hours). All groups except controls presented a significant reduction in uCa and uMg. Orlistat induced a significant increase in AP (CaOx) index(rat) compared, respectively, to baseline and to the diet period in NaOx (4.52 +/- 2.34 mg/24 hours vs. 0.94 +/- 0.86 and 1.53 +/- 0.93 mg/24 hours) and NaOx + soy oil (6.49 +/- 4.03 mg/24 hours vs. 0.54 +/- 0.17 and 1.76 +/- 1.32 mg/24 hours). CONCLUSION: These data suggest that the use of lipase inhibitors, especially under a diet rich in oxalate alone or associated with fat, leads to a significant and marked increase in urinary oxalate and a slight reduction in uCa and uMg that, taken together, resulted in an increase in AP (CaOx) index(rat), elevating the risk of stone formation.

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